Search Results

You are looking at 1-10 of 168

Figure 3; Values of PaO2 and PaCO2 measured in the arterial blood of the stressed group before and after the stress period. **P < .01 when compared with the rest period. The sample size for each group is 18.

K. C. Barcelos,

D. P. Furtado,

J. C. Ramacciato,

A. M. Cabral, and

D. A. Haas

Figure 3

Values of PaO₂ and PaCO₂ measured in the arterial blood of the stressed group before and after the stress period. **P < .01 when compared with the rest period. The sample size for each group is 18.

No Shortage of Stress

Kyle J. Kramer DDS, MS

Article Category: Editorial

Volume/Issue: Volume 69: Issue 4

Online Publication Date: Dec 19, 2022

DOI: 10.2344/anpr-69-04-07

Page Range: 1 – 2

Download PDF

Salivary Cortisol, Salivary Alpha Amylase, and the Dental Anxiety Scale

Hana Sadi DMD,

Matthew Finkelman PhD, and

Morton Rosenberg DMD

Article Category: Other

Volume/Issue: Volume 60: Issue 2

Online Publication Date: Jan 01, 2013

DOI: 10.2344/0003-3006-60.2.46

Page Range: 46 – 53

than men, and there is a tendency for the younger age groups to have more anxiety. 12 Dental anxiety was found to have a direct relationship with pain perception. 13 Rhudy and Meagher 14 suggested that the pain reactivity is modulated by emotional stress. In addition, Loggia et al 15 revealed changes in pain pathways on neuroimaging techniques with a negative emotional state. Furthermore, Klages et al 16 revealed that anxiety increases expected or experienced pain where patients with higher anxiety levels predicted a higher pain experience

Download PDF

Figure 1; Influences provided by vasodilators. Cardiac output is the principal determinant of systolic pressure while arterial resistance determines diastolic pressure. Venous dilation reduces venous return and subsequent cardiac output, while arterial dilation reduces arterial resistance. This accounts for the value of vasodilation in managing hypertension. In contrast, benefits for managing coronary artery disease (CAD) and congestive heart failure (CHF) relate to reducing stresses on the heart—preload and afterload. Preload (venous return) is a stress factor during diastole, and afterload (arterial resistance) stresses the heart during systole.

Daniel E. Becker

Figure 1

Influences provided by vasodilators. Cardiac output is the principal determinant of systolic pressure while arterial resistance determines diastolic pressure. Venous dilation reduces venous return and subsequent cardiac output, while arterial dilation reduces arterial resistance. This accounts for the value of vasodilation in managing hypertension. In contrast, benefits for managing coronary artery disease (CAD) and congestive heart failure (CHF) relate to reducing stresses on the heart—preload and afterload. Preload (venous return) is a stress factor during diastole, and afterload (arterial resistance) stresses the heart during systole.

Figure 1; Time course (latency) between the beginning of the IV infusion of lidocaine 2% or articaine 4% and the first tonic-clonic contraction in control and stressed rats. Values represent the means ± SEM. *P < .05 when compared with the respective control group; #P < .05 versus lidocaine control; and §P < .05 versus lidocaine stressed groups. The sample size for each group is shown in parentheses.

K. C. Barcelos,

D. P. Furtado,

J. C. Ramacciato,

A. M. Cabral, and

D. A. Haas

Figure 1

Time course (latency) between the beginning of the IV infusion of lidocaine 2% or articaine 4% and the first tonic-clonic contraction in control and stressed rats. Values represent the means ± SEM. *P < .05 when compared with the respective control group; #P < .05 versus lidocaine control; and §P < .05 versus lidocaine stressed groups. The sample size for each group is shown in parentheses.

Figure 2; Dose (IV infusion) of lidocaine or articaine to induce tonic-clonic seizure in controls and stressed rats. Values represent the means ± SEM. *P < .05 when compared with the respective control group. The sample size for each group is shown in parentheses.

K. C. Barcelos,

D. P. Furtado,

J. C. Ramacciato,

A. M. Cabral, and

D. A. Haas

Figure 2

Dose (IV infusion) of lidocaine or articaine to induce tonic-clonic seizure in controls and stressed rats. Values represent the means ± SEM. *P < .05 when compared with the respective control group. The sample size for each group is shown in parentheses.

Figure 1. ; Basic demographics of sample: gender (dark gray = M, light gray = F), race (dark gray = white, light gray = nonwhite), traumatic experience (dark gray = no , light gray = yes), pain (dark gray = no, light gray = yes).

Hana Sadi,

Matthew Finkelman, and

Morton Rosenberg

Figure 1.

Basic demographics of sample: gender (dark gray = M, light gray = F), race (dark gray = white, light gray = nonwhite), traumatic experience (dark gray = no , light gray = yes), pain (dark gray = no, light gray = yes).